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A positive regulatory feedback loop is established through direct binding of STAT3 to the AKR1C3 promoter (Zhou et al., 2021b)
Histological alterations and molecular mechanisms such as oxidative stress, induction of ferroptosis, and impaired mitochondrial function offer deeper insight into the pathogenesis of plastic-induced lung injury
Decreased GSH concentrations were noted in patients with alcoholic (2.55 0.1 mol/g of liver) and nonalcoholic liver diseases (2.77 0.1 mol/g of liver) compared with controls (4.14 0.1 mol/g of liver) that were speculated to be a contributing factor in liver injury and could enhance the risk of hepatic toxicity from a variety of toxic agents
However, an equimolar dose of dithiothreitol failed to increase GSH levels and protect mucosa from TNBS+ethanol-induced injury
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