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However, it is disappointing that IGF-1 has not been shown to play a very significant role in promoting tendon healing in clinical trials, which could reflect our poor understanding of the molecular mechanisms by which IGF-1 is involved in promoting tendon regenerative healing

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The sites on IRS-1 seen to be phosphorylated by chronic hyperglycemic and hyperinsulinemic conditions (S636/S639) are known to be substrates of mTORC1