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Oxidative stress has been increased as a result of mitochondrial dysfunction, endothelial nitric oxide synthase (eNOS) uncoupling, resulting in endothelial dysfunction, the aberrant activity of NADPH oxidases (NOX), suppressed activity of antioxidant enzymes and low molecular weight antioxidants (Galvan et al

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Sorafenib treatment significantly inhibited the growth of Tamoxifen resistant MCF-7 and ZR-75-1 cells in vitro (Fig

They say them because they hope things will improve