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The therapy rapidly restores B12 levels, often showing improvements in energy and cognitive function within 48 hours

The disruption of the ubiquitination of NRF2 allows the escape from the proteasomal degradation, activating the NRF2 transcriptional program (Dinkova-Kostova et al

\n\n\n\n The compounds multimechanistic nature also makes it challenging to evaluate through conventional clinical trial frameworks, which are typically designed around single-mechanism drugs with well-defined target receptors and predictable dose-response relationships

Current clinical guidance recommends restarting at the lowest dose when switching medications