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Abstract Mitochondrial defects result in dysregulation of metabolomics and energy homeostasis that are detected in upper motor neurons (UMNs) with TDP-43 pathology, a pathology that is predominantly present in both familial and sporadic cases of amyotrophic lateral sclerosis (ALS)

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Members access detailed reconstitution charts, personalized protocol builders, and expert-reviewed dosing guidance that accounts for individual factors most free resources ignore