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Numerous studies have confirmed the critical importance of irregular HGF/c-MET signaling in promoting growth, invasion, metastasis, apoptosis resistance, initiation of epithelial-mesenchymal transition (EMT), and enhancement of angiogenesis [7,8,9,10] (Fig

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Studies demonstrate its ability to stimulate cell migration and tendon fibroblast activity, leading to improved tendon outgrowth and repair, particularly in injuries like Achilles tendon ruptures (2)

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