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Conclusion and prospects The occurrence of ferroptosis can be summarized as disruption of iron metabolic homeostasis or System X c /GSH/GPX4 pathway by various factors causing the depletion of cellular antioxidants and amplification of ROS production, ultimately leading to the accumulation of lipid peroxides and triggering ferroptosis

Secondly, the ROS attack accelerates apoptosis of retinal tissues and cells and aggravates DR, 68 with the activated polyol pathway, hexosamine pathway, and PKC pathway, as well as advanced glycation end products (AGEs) accumulation involved in the process

It also supports wound healing, the integrity of the skin and helps prevent bruising

It works by influencing neurotrophic factors like BDNF and modulating dopamine and serotonin pathways, potentially supporting cognitive performance and neuroprotection in research settings