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Counteracting action of curcumin on high glucose-induced chemoresistance in hepatic carcinoma cells

A nonexclusive alternative explanation for the kinetic patterns with HEDS (and the decreased enzyme activity at high GSH concentrations) could be a Grx(SSCH 2 CH 2 OH) intermediate that is attacked by GSH according to monothiol mechanism (i) from Fig

They do not identify a particular toxin, metal or detoxification pathway by themselves, but the combination may justify evaluating toxic overload and depression together when the exposure history is credible

10B, exposure to SMG resulted in a significant increase in NO production in the zebrafish larvae, exhibiting a 4.8-fold elevation compared to control groups