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Thus, by promoting intimal hyperplasia, these vascular disorders (which are related to smooth muscle cell proliferation, collagen accumulation, and the extracellular matrix modification that may result from homocysteinylation of the vascular wall structure leading to connective tissue deterioration) might contribute to the atherogenic tendency of hyperhomocysteinemia
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In experimental research settings, this peptide combination is studied in non-clinical models focusing on peptide signaling behavior, molecular interaction pathways, and cellular response mechanisms under strictly controlled scientific conditions
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