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Although P-gp may be transiently upregulated in the acute phase, sustained injury ultimately suppresses its expression and function, leading to the failure of P-gpmediated efflux and the accumulation of neurotoxic compounds in the brain (160)

As highlighted in Table 2, IGF-1 peptide analogs are effective because of their specificity, small molecular size (0.61.2 kDa) and low toxicity [215219]

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