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Abstract Objectives: Hepatotoxicity is a severe outcome of methotrexate (MTX) therapy, limiting its clinical use and contributing to its related morbidity and mortality

The inflammatory response is intricately linked to redox balance, with ROS serving as key pro-inflammatory mediators

accordingly, when a supramaximal dose (1 M) of exendin-4 (to fully activate remaining surface receptors) was added in addition to IBMX after prior incubation with each test agonist, a linear relationship between internalization and cAMP response was restored (Fig

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