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GLP-1 in higher amounts also interacts with the parts of the brain that reduce appetite and signal a feeling of fullness.

Results from a blood-oxygen dependent functional MRI study [44] also suggest that mesolimbic reward-related areas may also mediate the anorectic effects of Ex in humans, with a hypothesised mechanism of decreasing responsiveness to contextual food cues (incentive salience, wanting) and increasing activity that correlates with consummatory reward value (liking) [45]

Indeed, GSH levels are markedly decreased in the SN of PD patients, which exacerbates oxidative injury and promotes ferroptosis (93)

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