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We have shown the generality of the combinatorial ubiquitin code with other TRIP12 substrates, suggesting that certain OTUD5-protected substrates are also regulated by DUB-resistant K29-linked ubiquitin chains

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Considering the effects of TNF on L cells and data from animal models, indicating that impaired glucose tolerance following high-fat diet-induced obesity can be ameliorated by anti-TNF therapy (95), one might expect a significant impact of anti-TNF treatment on glucose metabolism

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