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Microbiota-derived metabolites as drivers of gutbrain communication

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Furthermore, an attempt to develop a hypothesis that HHcy is a marker of oxidative and redox stress, as a result of an endogenous folate shuttle, which results in a relative endogenous endothelial folate deficiency due to a folate shuttle phenomenon to preferentially run the endothelial nitric oxide synthase (eNOS) reaction will be explored