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Description

Proposed mechanisms include upregulation of growth-hormone-receptor expression in injured tissue, VEGF-driven angiogenesis, modulation of the nitric-oxide pathway in vascular endothelium, and activation of FAK-paxillin signaling

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The standard medical approach starts at 0.25mg weekly and gradually increases over months

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In the adherent Rel3 cells treated with SU11274 we noticed increased expression of MUSASHI-1, which is considered as a marker of cancer stem cells (unpublished data)

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