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At low concentrations, it increases MMP1 and MMP2, which break down damaged or disorganized collagen and elastin
Moreover, a cell-by-cell analysis revealed that A-anticalin treatment reduced neuronal activity levels in all neurons ( Fig
Among them are several small-molecule compounds that have been screened using metabolomics, but their findings have not been confirmed in subsequent research, and many of the current animal and cell experiments have remained in the laboratory
Final remarks The high dependence of renal function on mitochondrial homeostasis, together with the central role of this organelle in cellular signaling, makes mitochondrial impairment a key in renal disease development
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