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However, in 2006 it became clear that GIP(3-42) does not function as a physiological antagonist in vivo as its maximal circulating levels are insufficient to elicit antagonistic effects (48)

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Mobile phases consisted of Solvent A (60% ACN, 40% HO, 10 mM Ammonium Formate with 0.1% Formic Acid) and Solvent B (90% IPA, 10% ACN, 10 mM Ammonium Formate with 0.1% Formic Acid)

This corresponds to product concentrations that enable reliable fibroblast stimulation without excess