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Therefore, early intervention in replenishing mGSH stores by supplying GSH precursor such as N -acetylcysteine (NAC) or targeting upstream mechanisms involved in mitochondrial dysfunction and ROS generation, such as the sustained activation of c-Jun N-terminal kinases (JNK), can result in the protection of APAP-mediated hepatotoxicity

YZ: Investigation, Writing original draft

Front Neurosci 12:1019

To estimate how the peptide levels during the first 12 months were associated with the fasting C-peptide/glucose ratio at 24 months, a linear slope for each peptide and each individual over the first four visits was calculated using linear regression models