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Simultaneously, there are disruptions in the expression of signaling pathways such as Transforming growth factor- (TGF-) signaling pathways, VEGF/VEGFR signaling pathways, Angiopoietin (Ang)/Tie signaling pathways, among others, ultimately driving glomerular fibrosis and progressing towards end-stage pathological conditions like glomerulosclerosis

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It activates the natural growth hormone secretagogue receptor (GHSR) in the brain, which is the same receptor that ghrelin binds to

YXS improved energy metabolism through increasing p-AMPK, PGC-1, GLUT4, and HIF-1 expression (Wu et al., 2020b)