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It is proposed to work by binding GHRH receptors on pituitary somatotroph cells, prompting the pituitary to synthesize and release its own growth hormone in a pulsatile pattern, with the body's somatostatin feedback loop still intact so the effect is self-limiting rather than a constant hormone flood

On the other hand, treatments with exogenous SA resulted in increasing cellular GSH levels and elevated activities of glutathione reductase and glutathione S-transferase enzymes, along with enhanced virus resistance (Fodor et al., 1997

TBI does not just kill neurons