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FIGURE 3 5 Conclusion, challenges and outlook This review highlights the pivotal role of ferroptosis in PF and emphasizes the importance of identifying ferroptosis-related therapeutic targets, such as iron metabolism, ROS production, PUFAs metabolism, and the antioxidant system, as key pathways for therapeutic intervention

Cop1 constitutively regulates c-Jun protein stability and functions as a tumor suppressor in mice

It is typically reserved for autoimmune conditions with significant accumulated tissue damage

It donates an electron to the harmful substance, neutralizing it and turning it into a less harmful compound