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Accordingly, the areas of unmet need for new treatments are reflected in the lesions that underly the hyperglycaemia, notably impaired function and number of pancreatic beta cells, insulin resistance and glucose toxicity, which are frequently aggravated by excess adiposity, abnormalities of glucagon secretion, an altered incretin response and disturbances in lipid metabolism [6]

Takasu T, Takakura S (2019) Effect of ipragliflozin, an SGLT2 inhibitor, on cardiac histopathological changes in a non-diabetic rat model of cardiomyopathy

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