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In experimental models of Alzheimer's disease, GLP-1 receptor agonists have shown promise in reducing amyloid-beta plaque formation and tau protein phosphorylation, two hallmark pathological features of the condition

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The immune system hypothesis includes the alteration of the proper functioning of the central nervous system (CNS) associated with an abnormal response to common antigens, stimulation of the inflammatory and cell-mediated immune responses, and upstream regulation of oxidative and nitrosative pathways, and autoimmune responses against neuronal and other cells (Marshall-Gradisnik et al