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This, in turn, stimulates SREBP2 activity via integrin alpha V-mediated activation of the PI3K/Akt pathway, and enhances the activation of HMGCR

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These findings reveal that metabolic abnormalities may involve in the pathophysiological mechanism of preeclampsia, which suggests that in the future researches, more specific metabolic pathways need to be explored in preeclampsia based on animals and in vitro models, which is of great significance to the development of new metabolic drugs for the alleviation of preeclamptic symptoms