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Anti-inflammatory microglia contributes to stroke recovery by expressing anti-inflammatory cytokines, such as IL-10, IL-4, and some neurotrophic factors which prevent inflammation (100) and play important roles in tissue repair and wound healing ( All of these recovery processes lead to a stabilization of the permeability of the BBB, although several animal studies have shown increased permeability up to 1 (68) and further (189), suggesting that BBB remains open and permeable through this subacute stage

For example, modest induction of acinar apoptosis in MAP may help control inflammation, whereas in SAP, cell necrosis should be prevented as much as possible