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This finding underscores the specificity of the neural circuit regulation of "vagal afferent fibersNTS GLP1 ARC" by EA, and it also further enriches the multi-target mechanism of EA in improving obesity

Third, as we will detail below (see Potential Pathogenic Mechanisms of Hyperhomocysteinemia), our own work (Durand et al, 1997a) and the work of Lentz et al (1996) and Bellamy et al (1998) give experimental evidence that moderate hyperhomocysteinemia, which leads to vascular dysfunction, might increase the cardiovascular risk independently of other risk factors