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Journal of Endocrinological Investigation, 47(3), 567-578

Notably, Lpin1 induction is dependent on elevated ROS and ATM levels, and this process is inhibited by the N-acetyl cysteine antioxidant (66)

For disrupting tumor glucose metabolism, nanomaterials share merits in active targeting via surface modifications, improving drug accumulation within the tumor, on-demand drug release through stimuli-responsive designs according to endogenous stimulating signals, such as pH, ROS, and enzymes, and multifunctionality integrating diagnostics with combination therapies [33,34,35]

Individual results may vary, and the safety of BPC-157 for human use has not been established through clinical trials.