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However, short-term acute changes in glucose concentrations may not directly contribute to the synaptic plasticity associated with diabetes, unless extremely severe.132 Glutamate receptors, including the amino-3-hydroxy-5-methyl-4-isoxazolepro-pionicacid (AMPA) and N-methyl-D-aspartate (NMDA) receptors, mediate excitatory synaptic transmission in the CNS, and its expression in the postsynaptic membrane is associated with LTP and LTD and is involved in the regulation of learning and memory activities

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8a), indicating that while the Cagri-CTR complex can sample this conformation, it occurs at lower frequency than rAmy-CTR