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They activate GLP-1 receptors, promoting insulin secretion while also inhibiting glucagon secretion from the pancreas

Remarkably, they are not recognized as foreign bodies, allowing them to effortlessly enter the bloodstream without any loss

In parallel, synthetic biology approaches explore LR3 analogs with modified IGF-1R affinity, increased selectivity versus the insulin receptor, or biased activation of specific signaling pathways (pMAPK versus pAkt) to dissociate proliferative from metabolic effects

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