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These organs are all therefore potential sites of alerted signalling if endogenous fetal GLP-1 levels are altered, or if GLP-1 signalling is over-activated by exposure to GLP1RAs during fetal development

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The main mechanism of action of SGLT2is is the inhibition of glucose reabsorption in the kidneys, leading to increased glucose excretion in the urine and a direct reduction in blood glucose levels

The possibility that DPP4i may be harmful to animals with immune deficiency still needs to be clarified