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Thummel et al

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Interestingly, reductions in bodyweight and HbA1 c were similar in patients with improvements in liver histology (responders) and those without (non-responders), suggesting that, beyond bodyweight reduction and improvement in glucose control, other mechanisms may be involved in the beneficial effects of liraglutide

In mouse, a NOD.H-2h4 strain prone to AIT and susceptible individuals, excess I increases the number of intrathyroidal infiltrating Th17 and inhibits the development of Treg, causes abnormal expression of tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) in thyrocytes, leading to their apoptosis (44, 45)