Activation of the pY397 autophosphorylation site of FAK promotes SRC binding, leading to the conformational activation of SRC, and, subsequently, a dual activated FAKSRC signalling complex (Mitra et al, 2005)
It is not merely a byproduct of rapid tumor growth but a dynamic driver of metabolic and molecular reprogramming
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For instance, our group recently showed that a mutation in the ribosomal RPL10 gene, known as RPL10 R98S, enhances serine/glycine synthesis in T-cell leukaemia by increasing both PSPH transcriptional and translational levels (3)
