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Oleoylethanolamide (OEA), an ethanolamide fatty acid synthesized in the body, can regulate glucose homeostasis and weight loss by stimulating GLP-1 secretion from intestinal L-cells and insulin secretion from -cells in a GPR119-dependent manner and by affecting the abundance of the bacterium Akkermansia muciniphila [146, 147]

Oxidative stress can disrupt these tolerance mechanisms by inducing apoptosis of regulatory T cells (Tregs), which are critical for maintaining immune homeostasis and preventing autoimmunity [141]

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