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The three non-coded amino acid substitutions serve critical functions: Aib2 provides stability against dipeptidyl peptidase-4 degradation, Aib20 optimizes GIP receptor activity and pharmacokinetic properties, and alpha-methyl-L-leucine at position 13 enhances receptor binding selectivity[2]

Promotion of NLRP3 activity by oxidative stress is likely to play a prominent role in a wide range of poorly understood inflammation-related conditions, such as myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), long COVID, Alzheimers disease, and other disorders associated with aging