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Mechanistically, the inflammatory response after heart transplantation is mediated by TLR4/TRIF signaling pathway, which facilitates neutrophil migration to coronary endothelial cells, resulting in ferroptosis in the transplant endothelial cells

Thus, here, we showed that exogenous glutathione has the potential to restore the redox status of cardiac mitochondria due to the reduction of oxidative stress, improvement of mtNOS activity and H 2 S levels which leads to the inhibition of mPTP and improvement of the functions of the cardiovascular system in old rats

Lazarus M, Kashyap AK, Borkar RS, Ajmariya M

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