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Vascular edema (accumulation of extracellular water due to increased blood-brain barrier permeability) and cytotoxic edema (disruption of Na+/K + pump function on the cell membrane) are considered the underlying mechanisms of HACE ( The pathophysiology of high altitude illnesses encompasses limited oxygen supply, hypoxia-induced physiological adaptations and compensatory responses, changes in blood rheology, and the development of related tissue damage and complications

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2011), and orchestrates the FAK-paxillin signaling cascade governing cell migration within tendon tissue