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To test this, we investigated the vulnerability to ferroptosis of aging nematodes upon the natural loss of glutathione during lifespan

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58 A further source of oxidative damage can originate from activated peripheral macrophages or brain resident glial cells in response to microenvironmental activators, such as A and -synuclein protein as well as circulating cytokines, all of which are observed in the setting of neurodegenerative disease