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Prolonged iron overload promotes sustained development of endometriosis, PCOS, and ectopic endometrial lesions, which may directly affect oocyte and embryonic development

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Ovarian tumours epigenetically reprogram resident MSCs toward a partial mesenchymal-to-epithelial transition controlled by WT1/EZH2, enabling tight CA-MSCcancer cell binding and co-metastasis. Pharmacological EZH2 inhibition attenuated this metastatic escort function, yet did not abolish it, indicating that even modified MSCs remain metastasis-competent [199]

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