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Kla modification activates the promoter region of the PD-L1 gene, and this epigenetic regulation provides a molecular basis for high PD-L1 expression, further inhibiting the antitumor activity of CD8 + T cells (94, 95)
The work was supported by grants from the National Natural Science Foundation of China (11572064, 31971242, and 31771599), the National Key Technology R&D Program of China (2016YFC1102305), and Chongqing Science and Technology Bureau (cstc2019kjrc)
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