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Together, the IGF-1/IGF-1R/ hsa-let-7c axis can control the odonto/osteogenic differentiation of IGF-1-treated SCAPs via the regulation of JNK and p38 MAPK signaling pathways

Therefore, selenium transport is part of a dynamic regulatory network that integrates redox signaling, cellular metabolism, and intestinal barrier function [2, 90]

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These are different pharmacological endpoints citing one to support the other misrepresents what the studies actually measured