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Exenatide activated eNOS and NO production in endothelial cells, induced dose-dependent vasorelaxation, and mitigated high-glucose or lipid-induced endothelial dysfunction in arterioles ex vivo, effects which were reduced by AMPK (AMP-activated protein kinase) inhibition [86]

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AHR assessment using the FinePointe RC system (Buxco Research Systems), pulmonary ILC2 and BAL fluid cell analysis, cytokine measurement in BAL supernatant, and lung histological examination were performed according to the established protocols (46, 48, 71, 74)

For instance, the specific EPA to DHA ratio in EFAs, the bioavailability of different forms of zinc, and the potential utilization of innovative delivery systems like liposomes and nanoparticles warrant thorough investigation