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In particular, one investigation involved normal and dexamethasone-induced catabolic murine models

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These findings thus support a new conceptual framework for targeting GLP1R for prostate cancer benefit, including 1) a potential new therapeutic target for NEPC, 2) a novel context for Gi-GPCR signaling downstream of GLP1R, and 3) insight into shared signaling mediators downstream of Notch and GLP1R signaling in prostate cancer lineage plasticity