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While the first studies showing this were in mice [14], human studies have corroborated these findings [200], although its importance in relationship to traditional clearance pathways across the BBB and subarachnoidal granulations has been challenging to quantify

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The goal is to recognize those biological changes that underlie aberrant epigenetic programming of brain plasticity, and to recognize mitochondrial signaling pathways, metabolic factors, transcriptomic profiles and structural changes that indicate flexible adaptability or the lack thereof