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Activating GPX4 can significantly reduce ferroptosis in intestinal epithelial cells (IECs) and improve IBD symptoms ( The role of ferroptosis in the pathogenesis of IBD involves multiple pathways, including iron metabolism disorders, intestinal epithelial barrier damage, genetic susceptibility, immune dysregulation, and gut microbiota imbalance

Specifically, MUFAs react with coenzyme A (CoA) under the catalysis of ACSL3 to form the intermediate product MUFA-CoA, which is inserted into the fatty acid chain of phospholipids

Cigarette smoke (CS) is another potential risk factor for the ferroptosis of CECs

The international glossary on infertility and fertility care, 2017