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Although a defined mechanistic evaluation of microbe-dependent pathology of AD-dementia is yet to be uncovered, previous studies indicate that AD pathogenesis is perhaps associated with age-dependent depletion of healthy microbes with concurrently increased burden of infectious opportune, enterotoxigenic bacteria and viruses, i.e., Bacteroides fragilis and herpes simplex virus-1 (HSV-1) (Lukiw et al., 2021)

Additionally, GLP-1 RAs may influence other epigenetic regulators, such as sirtuins and histone deacetylases, which play a role in crucial cellular functions like inflammation and stress response

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A., et al (2020)