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This neurotoxic effect can begin via prenatal exposure, with current evidence reporting prenatal exposure to PAH to be associated with several neurodevelopmental and neurocognitive disorders such as attention deficit hyperactivity disorder (ADHD), autism, anxiety, adolescent behavioral problems, depression and school performance

Upon inhalation, PAHs are bioactivated by cytochrome P450 (CYP450) enzymes, converting them into highly reactive electrophiles such as diol-epoxides and redox-active quinones

To verify the functions of these overlapping genes, we conducted KEGG enrichment analysis and found that these genes were significantly enriched in the VEGF signaling pathway, FoxO signaling pathway, MAPK signaling pathway, mTOR signaling pathway, and Wnt signaling pathway (Figure 1B)

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