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Losing weight rapidly can also mean losing muscle and potentially bone, particularly without sufficient protein, strength training, and clinical monitoring

Figure 1 3.1 Insulin resistance and -cell secretion defects At the molecular level, peripheral insulin resistance arises from impaired insulin receptor substrate (IRS-1) phosphorylation and downstream PI3K/Akt signaling in skeletal muscle and adipose tissue, combined with failure to suppress hepatic glucose production via the gluconeogenic pathway (3436)

Time constraints or physical limitations might make it difficult to exercise

Both are injectable peptides, but they work through entirely different biological pathways