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Portal Hypertension and Sinusoidal Biology In established cirrhosis models (CCl 812 weeks), portal hypertension develops from intrahepatic vascular resistance due to sinusoidal HSC contraction, capillarisation (loss of endothelial fenestrations and CD32b LSEC marker), and mechanical fibrosis-driven resistance
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Professional medical consultation is advised before starting CJC-1295
tremor, tachycardia) Asthma/COPD: 2 blockade may precipitate bronchospasm , especially with non-selective agents Peripheral arterial disease: Reduced 2-mediated vasodilation may worsen claudication Psoriasis: beta blockers can exacerbate pre-existing psoriasis Adverse Effects Adverse effects reflect blockade of receptors in non-cardiac tissues and include the following: Bronchospasm (2 blockade) Bradycardia Cold extremities and Raynauds phenomenon Hypotension Fatigue and dizziness Sleep disturbance (lipophilic drugs) including nightmares Erectile dysfunction Interactions Beta-blockers have several important drug interactions that may affect respiratory and cardiovascular function
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