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, TLRs) expressed on the inflammatory M initiate the innate immune signaling pathways and trigger a variety of pro-inflammatory cytokines production, which in turn instructs a pro-inflammatory adaptive immune response involving primarily Th1 cells (204, 205)

Accelerated metabolism and exclusion of 4-hydroxynonenal through induction of RLIP76 and hGST5.8 is an early adaptive response of cells to heat and oxidative stress

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