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Consequently, the metabolic pathways initiated by G proteins within GLP-1R involve cAMP activation, extracellular signal-regulated kinases 1 and 2 (ERK 1/2) and increased intracellular calcium levels (17, 19)

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NAE reports associated with GLP-1 RAs were extracted, and disproportionality analysis was performed to identify NAEs significantly disproportionately related to these agents, which were defined as GLP-1 RA-related NAEs in this work